Typically the parenchymal cysts are small (1cm) whereas the subarachnoid ones can be much bigger (up to 9cm): differential therefore being arachnoid cyst. It is the most common cause of epilepsy in endemic areas (South-east Asia, South America) with progression through the 4 stages taking anywhere between 1 to 9 years.> Radiographic features: Imaging findings depend on location and stage of infection.> Subarachnoid-intraventricular: When in the subarachnoid space/intraventricular, the the cysts typically do not have a visible scolex. In the basal cisterns they can be grape-like (racemose). The cysts are typically 1-2 cm in diameter 2. Usually the cysts are similar in signal intensity to CSF, although occasionally cyst fluid may somewhat differ.> In the ventricles there is often (79%) 2 associated ventriculitis often leading to aqueductal stenosis and hydrocephalus.> Parenchymal: Parenchymal cysts usually involve the grey white matter junction.> Vesicular: cyst with dot sign; CSF density/intensity; hyperintense scolex on T1 can sometimes be seen; no enhancement is typical, although very faint enhancement of the wall and enhancement of the scolex may be seen> Colloidal vesicular: cyst fluid becomes turbid CT: hyperdense to CSF; MRI T1: hyperintense to CSF; surrounding oedema; cyst and the wall becomes thickened and brightly enhances scolex can often still be seen as an eccentric focus of enhancement> Granular nodular: oedema decreases; cyst retracts; enhancement persists but is less marked>Nodular calcified: Endstage quiescent calcified cyst remnant, no oedema; no enhancement on CT; signal drop out on T2 and T2* sequences; some intrinsic high T1 signal may be present; long term enhancement may be evident on MRI, and may predict ongoing seizures.">