**Core Concept**
Uveoscleral outflow is one of the two main pathways for aqueous humor drainage from the eye, the other being trabecular meshwork outflow. The uveoscleral pathway involves the active transport of aqueous humor through the ciliary muscle and sclera into the suprachoroidal space.

**Why the Correct Answer is Right**
The correct answer is related to the use of **prostaglandin analogs**, which increase uveoscleral outflow by stimulating the production of prostaglandin E2 (PGE2) in the ciliary body. PGE2 causes the contraction of the ciliary muscle, which in turn opens the uveoscleral pathway and increases aqueous humor drainage. This mechanism is the basis for the use of prostaglandin analogs in the treatment of glaucoma.

**Why Each Wrong Option is Incorrect**
* **Option A:** This option is incorrect because **beta-blockers** primarily decrease aqueous humor production through the inhibition of beta-adrenergic receptors in the ciliary body.
* **Option B:** This option is incorrect because **carbonic anhydrase inhibitors** primarily decrease aqueous humor production through the inhibition of carbonic anhydrase in the ciliary body.
* **Option C:** This option is incorrect because **miotics** primarily increase trabecular meshwork outflow by contracting the ciliary muscle and increasing the surface area for aqueous humor drainage.

**Clinical Pearl / High-Yield Fact**
Prostaglandin analogs are a class of medications that are particularly useful in the treatment of glaucoma because they can increase uveoscleral outflow without affecting intraocular pressure (IOP) in the short term. This makes them a useful option for patients who are sensitive to the side effects of other glaucoma medications.

**Correct Answer: D. Prostaglandin analogs**