**Core Concept**
Stimulation of lung C fiber endings is a critical event in the pathophysiology of various respiratory diseases, including asthma and chronic obstructive pulmonary disease (COPD). These C fibers are non-myelinated afferent nerve endings that respond to changes in lung inflation, ventilation, and chemical composition.

**Why the Correct Answer is Right**
Stimulation of lung C fiber endings typically leads to the release of neurotransmitters, such as substance P and calcitonin gene-related peptide (CGRP), which cause vasodilation and increased vascular permeability. This results in the symptoms of bronchospasm, cough, and wheezing. Furthermore, C fiber stimulation also activates the juxta-alveolar interstitial cells, which release inflammatory mediators and contribute to airway inflammation. The correct answer is the option that does not describe a known consequence of C fiber stimulation.

**Why Each Wrong Option is Incorrect**
**Option A:** Vasodilation is a known consequence of C fiber stimulation, as it leads to increased blood flow to the lung tissue.
**Option B:** Bronchospasm is also a result of C fiber stimulation, as it activates the release of neurotransmitters that cause smooth muscle contraction in the airways.
**Option C:** Airway inflammation is a consequence of C fiber stimulation, as it activates the juxta-alveolar interstitial cells to release inflammatory mediators.

**Clinical Pearl / High-Yield Fact**
It is essential to remember that C fiber stimulation is a key event in the pathophysiology of various respiratory diseases, and targeting these fibers can be a potential therapeutic strategy for managing conditions such as asthma and COPD.

**Correct Answer: C. Inhibition of vasodilation is not a consequence of stimulation of lung C fiber endings.**