**Core Concept**
Post-streptococcal glomerulonephritis (PSGN) is an immune complex-mediated glomerulonephritis that occurs following a streptococcal infection, typically after a pharyngitis or skin infection. The pathogenesis involves the deposition of immune complexes containing antigens from the streptococcal bacteria and antibodies produced by the host.

**Why the Correct Answer is Right**
The correct answer is associated with PSGN because of the role of streptococcal antigens in triggering the immune response. The deposition of immune complexes containing these antigens in the glomeruli leads to an inflammatory response, characterized by the activation of complement, release of cytokines, and influx of neutrophils. This results in damage to the glomerular capillaries, leading to the characteristic histological features of PSGN.

**Why Each Wrong Option is Incorrect**
* **Option A:** This option is incorrect because PSGN is not typically associated with a direct viral infection. While viral infections can trigger an immune response, the pathogenesis of PSGN is more closely linked to bacterial infections, specifically streptococcal infections.
* **Option B:** This option is incorrect because membranous nephropathy is a different type of glomerulonephritis characterized by the deposition of immune complexes on the subepithelial side of the glomerular basement membrane. It is not directly associated with streptococcal infections.
* **Option C:** This option is incorrect because minimal change disease is a type of nephrotic syndrome characterized by the loss of foot processes of podocytes. It is not typically associated with streptococcal infections or immune complex deposition.

**Clinical Pearl / High-Yield Fact**
PSGN typically presents 1-3 weeks after a streptococcal infection, with symptoms including hematuria, proteinuria, and hypertension. It is often associated with a history of recent streptococcal pharyngitis or skin infection.

**Correct Answer:** D.