**Core Concept**
Aspirin poisoning, also known as salicylate poisoning, is a toxic condition caused by excessive ingestion of aspirin or salicylates. It can lead to various systemic effects due to the inhibition of the enzyme cyclooxygenase (COX), resulting in the accumulation of salicylic acid in the body.

**Why the Correct Answer is Right**
Hypothermia is not typically associated with aspirin poisoning. In fact, patients often present with hyperthermia due to the uncoupling of oxidative phosphorylation in the mitochondria, leading to an increase in metabolic rate and heat production. This results in tachycardia, sweating, and fever, which are characteristic symptoms of aspirin poisoning.

**Why Each Wrong Option is Incorrect**
**Option A:** Acidosis is a true complication of aspirin poisoning. The uncoupling of oxidative phosphorylation leads to an increased production of lactate, resulting in metabolic acidosis.

**Option C:** Dehydration is also a true complication of aspirin poisoning. The increased renal excretion of water and electrolytes, along with the loss of gastric fluids, can lead to dehydration.

**Option D:** Oliguria is a true complication of aspirin poisoning. The renal vasoconstriction caused by the inhibition of prostaglandin synthesis can lead to a decrease in renal blood flow and glomerular filtration rate, resulting in oliguria.

**Clinical Pearl / High-Yield Fact**
In cases of aspirin poisoning, the presence of metabolic acidosis, tachycardia, and hyperthermia should prompt immediate treatment with activated charcoal, supportive care, and, if necessary, hemodialysis to remove the excess salicylates from the body.

**✓ Correct Answer: B. Hypothermia. Hypothermia is not typically associated with aspirin poisoning.**