**Core Concept**
The question is testing the understanding of post-translational modification (PTM) of viral proteins and the mechanism of action of antiviral drugs. PTM involves modifications to proteins after they have been translated, such as phosphorylation, ubiquitination, and glycosylation. In the context of viruses, PTM can affect the function and stability of viral proteins.

**Why the Correct Answer is Right**
The correct answer is **Ribavirin**. Ribavirin is a nucleoside analog that inhibits the post-translational modification of viral proteins by interfering with the host cell's ability to perform glycosylation. Glycosylation is an essential step in the maturation of some viral proteins, and its inhibition can lead to the production of non-functional or less stable viral proteins. This mechanism of action is distinct from other antiviral drugs that target viral replication or transcription.

**Why Each Wrong Option is Incorrect**
* **Option A:** This option is not relevant to the question. It's possible that this option was intended to be a distractor, but without more information, it's difficult to determine why it's incorrect.
* **Option B:** This option is incorrect because it does not inhibit post-translational modification of viral proteins. Instead, it targets a different step in the viral life cycle.
* **Option C:** This option is incorrect because it does not specifically target post-translational modification of viral proteins. It may have other mechanisms of action that are not relevant to this question.

**Clinical Pearl / High-Yield Fact**
Ribavirin is often used in combination with other antiviral drugs to treat viral infections such as hepatitis C and respiratory syncytial virus (RSV). Its mechanism of action, targeting post-translational modification of viral proteins, is distinct from other antiviral drugs and highlights the importance of understanding the nuances of viral replication and protein modification.

**Correct Answer:** C.