## Core Concept
Wilson's disease, also known as hepatolenticular degeneration, is a genetic disorder characterized by excessive accumulation of **copper** in the body, particularly in the **liver**, **brain**, and other vital organs. This accumulation is due to a mutation in the **ATP7B gene**, which plays a crucial role in regulating copper transport and excretion.

## Why the Correct Answer is Right
The correct answer involves understanding the primary sites of copper deposition in Wilson's disease. Copper accumulates in the **liver** due to impaired biliary excretion, leading to liver damage. It also accumulates in the **brain**, particularly in the **basal ganglia**, causing neurological symptoms. Additionally, copper deposits in the **cornea** of the eye, forming **Kayser-Fleischer rings**, which are a hallmark of the disease.

## Why Each Wrong Option is Incorrect
- **Option A:** While copper does deposit in various tissues, option A is not specified; however, based on the context, if it doesn't include liver, brain, or cornea, it's incorrect.
- **Option B:** Similarly, without specifics, if option B doesn't align with known sites of copper deposition in Wilson's disease, it's incorrect.
- **Option D:** This option is incorrect if it suggests sites not primarily associated with copper deposition in Wilson's disease.

## Clinical Pearl / High-Yield Fact
A key clinical pearl is that **Kayser-Fleischer rings**, which are copper deposits in the Descemet's membrane of the cornea, are a diagnostic feature of Wilson's disease. Their presence, especially in a patient with liver disease and neurological symptoms, is highly suggestive of the condition. Early diagnosis is critical for treatment and prevention of long-term damage.

## Correct Answer Line
**Correct Answer: C.**