**Core Concept**
Hurler syndrome is a genetic disorder caused by a deficiency of a specific enzyme involved in the breakdown of sugar molecules. This enzyme is crucial for the degradation of glycosaminoglycans, particularly dermatan sulfate and heparan sulfate. The deficiency leads to the accumulation of these molecules in the body.

**Why the Correct Answer is Right**
The correct answer is due to the deficiency of **alpha-L-iduronidase**, an enzyme responsible for breaking down glycosaminoglycans. Without this enzyme, dermatan sulfate and heparan sulfate accumulate in the body, leading to the clinical features of Hurler syndrome, including clouded corneas, deafness, and intellectual disability.

**Why Each Wrong Option is Incorrect**
**Option A:** This option is incorrect because the enzyme associated with Hurler syndrome is not related to this choice.
**Option B:** This option is also incorrect as it does not correspond to the enzyme deficient in Hurler syndrome.
**Option D:** Similarly, this option is incorrect as it is not the enzyme responsible for the condition.

**Clinical Pearl / High-Yield Fact**
Hurler syndrome is an **X-linked recessive disorder**, but it exhibits an autosomal recessive pattern of inheritance because the gene is located on chromosome 4, which is an autosome. This disorder is part of a group of conditions known as mucopolysaccharidoses.

**Correct Answer:** D. alpha-L-iduronidase