**Core Concept**
Haloperidol, a typical antipsychotic, can cause **extrapyramidal side effects (EPS)** due to its high affinity for D2 dopamine receptors in the striatum. EPS are a common adverse effect of typical antipsychotics, manifesting as a result of dopaminergic blockade in the nigrostriatal pathway.

**Why the Correct Answer is Right**
Haloperidol's EPS are primarily due to its blockade of D2 receptors in the striatum, which disrupts the balance between the dopaminergic and cholinergic systems. This leads to an overactivity of the cholinergic system, causing symptoms such as restlessness, fidgetiness, and irritability. The patient is experiencing **akathisia**, a type of EPS characterized by subjective feelings of restlessness and a compelling need to be in constant motion.

**Why Each Wrong Option is Incorrect**
**Option A:** Increasing the dose of haloperidol will not alleviate EPS, as it will only exacerbate the blockade of D2 receptors and worsen the symptoms.
**Option B:** Switching to a different antipsychotic medication may be beneficial, but it is not the most immediate solution to manage EPS. In this scenario, a specific treatment for EPS is required.
**Option C:** Risperidone, an atypical antipsychotic, may have a lower risk of EPS compared to haloperidol but is not the most appropriate treatment strategy for managing EPS in this patient.

**Clinical Pearl / High-Yield Fact**
The use of **benzhexol (trihexyphenidyl)** or **propranolol** can be an effective strategy for managing EPS, particularly akathisia, in patients taking typical antipsychotics like haloperidol.

**Correct Answer: B. Switching to a different antipsychotic medication with a lower risk of EPS, such as aripiprazole or olanzapine, or adding a medication specifically for EPS like benzhexol or propranolol.**