**Core Concept**
Cushing syndrome is a clinical condition caused by excessive exposure to glucocorticoids, leading to various systemic manifestations. The underlying pathophysiology involves the effects of cortisol on multiple organ systems, including the metabolic, cardiovascular, and musculoskeletal systems.

**Why the Correct Answer is Right**
Hypoglycemia is not typically associated with Cushing syndrome. In fact, patients with Cushing syndrome often present with hyperglycemia due to the glucocorticoid-induced increase in glucose production and insulin resistance. The correct answer is hypoglycemia because Cushing syndrome is characterized by an increase in glucose levels, not a decrease. The cortisol excess leads to an increase in gluconeogenesis, glycogenolysis, and insulin resistance, resulting in hyperglycemia.

**Why Each Wrong Option is Incorrect**
**Option B:** HT (Hypertension) is a common feature of Cushing syndrome due to the mineralocorticoid effects of cortisol, which leads to sodium retention and volume expansion.

**Option C:** Proximal myopathy is indeed a feature of Cushing syndrome, resulting from the muscle wasting effects of cortisol. The glucocorticoid-induced myopathy is a well-documented complication of long-term corticosteroid therapy.

**Option D:** Centripetal obesity, also known as truncal obesity, is a characteristic feature of Cushing syndrome. The cortisol excess leads to redistribution of fat to the central and upper body regions.

**Clinical Pearl / High-Yield Fact**
It's essential to remember that Cushing syndrome can be caused by both endogenous (e.g., pituitary adenoma) and exogenous (e.g., glucocorticoid therapy) sources of glucocorticoids.

**✓ Correct Answer: A. Hypoglycemia**