**Core Concept**
The presence of glycosuria (glucose in urine) in a patient with normal blood glucose levels is indicative of renal glucose transport dysfunction. The kidneys play a crucial role in glucose reabsorption, and any impairment in this process can lead to glycosuria.

**Why the Correct Answer is Right**
In a normal individual, glucose is reabsorbed from the filtrate in the renal proximal convoluted tubule (PCT) via the sodium-glucose cotransporter 2 (SGLT2). In patients with normal blood glucose levels but glycosuria, it suggests an issue with SGLT2 function or regulation. This can be due to various factors such as Fanconi syndrome, which involves impaired glucose reabsorption in the PCT. Another possibility is a rare genetic disorder known as renal glycosuria, where the kidneys are unable to reabsorb glucose properly due to a mutation in the SGLT2 gene.

**Why Each Wrong Option is Incorrect**
* **Option A:** This option is likely a distractor and may refer to a condition like diabetes mellitus, where high blood glucose levels would lead to glycosuria. However, the question specifies that the patient's blood glucose levels are within normal limits.
* **Option B:** This option could refer to a condition like hyperglycemia, which is not applicable in this scenario as the patient's blood glucose levels are normal.
* **Option C:** This option may refer to a condition like Fanconi syndrome, which involves impaired glucose reabsorption in the PCT, but it is not the most specific answer given the context of the question.

**Clinical Pearl / High-Yield Fact**
It's essential to remember that glycosuria in a patient with normal blood glucose levels can be an indicator of renal glucose transport dysfunction, which may be due to various underlying causes.

**Correct Answer: C. Fanconi syndrome**