**Core Concept**
The question is testing the understanding of oxygen toxicity in infants, specifically its impact on the retina. Oxygen toxicity, also known as retinopathy of prematurity (ROP), occurs when premature infants are exposed to high concentrations of oxygen, leading to abnormal blood vessel growth in the retina.

**Why the Correct Answer is Right**
In infants, particularly those born prematurely, the retinal blood vessels are still developing. When exposed to high levels of oxygen, these blood vessels can grow abnormally, leading to the formation of fragile, non-functional blood vessels. This abnormal growth can cause retinal detachment, scarring, and potentially lead to blindness. The pathophysiology involves the upregulation of vascular endothelial growth factor (VEGF) in response to the high oxygen levels, promoting the growth of new, but abnormal, blood vessels.

**Why Each Wrong Option is Incorrect**
**Option A:** This option is not relevant to the context of oxygen toxicity in infants.
**Option B:** This option is incorrect because it does not directly relate to the pathophysiology of oxygen toxicity in infants.
**Option C:** This option is not a recognized cause of blindness due to oxygen toxicity in infants.

**Clinical Pearl / High-Yield Fact**
Retinopathy of prematurity (ROP) is a leading cause of blindness in premature infants. The risk of ROP increases with the duration and concentration of oxygen therapy. Early detection and treatment can significantly improve outcomes.

**Correct Answer:** C. Retinopathy of Prematurity (ROP).