All may be seen in deep burns except
**Core Concept**
Deep burns, also known as third-degree burns, are characterized by full-thickness destruction of the skin and potentially underlying tissues. The extent of damage can lead to various systemic complications. In deep burns, the body's response to injury is altered, affecting the release of various hormones and inflammatory mediators.
**Why the Correct Answer is Right**
The correct answer is related to the pathophysiology of deep burns. Deep burns can lead to hypovolemic shock due to fluid loss, but they also stimulate the release of stress hormones, such as cortisol and epinephrine, which can cause hyperglycemia. In addition, the release of inflammatory mediators, like bradykinin and histamine, can lead to vasodilation and increased vascular permeability, resulting in edema. The correct answer, however, is related to a condition that is not typically seen in deep burns.
**Why Each Wrong Option is Incorrect**
**Option A:** Hyperkalemia - Hyperkalemia is more commonly seen in electrical burns or crush injuries, which can cause muscle cell lysis and release potassium ions into the bloodstream.
**Option B:** Hypocalcemia - Hypocalcemia is often seen in severe burns due to the deposition of calcium in burned tissues and the release of phosphorus from damaged cells, leading to a decrease in serum calcium levels.
**Option C:** Hypercalcemia - Hypercalcemia is not typically seen in deep burns. While it can occur in other conditions, such as malignancy or vitamin D intoxication, it is not a common complication of deep burns.
**Clinical Pearl / High-Yield Fact**
It's essential to remember that deep burns can lead to a complex interplay of systemic complications, including hyperglycemia, hypovolemic shock, and edema. Early recognition and management of these complications are crucial to prevent further morbidity and mortality.
**Correct Answer:** C. Hypercalcemia.