**Core Concept**
Alcoholic liver disease (ALD) encompasses a spectrum of liver damage caused by chronic alcohol consumption, ranging from steatosis (fatty liver) to cirrhosis. The pathophysiology involves the accumulation of fat in hepatocytes, oxidative stress, and inflammation, ultimately leading to fibrosis and cirrhosis.

**Why the Correct Answer is Right**
The correct answer, **D. All**, is correct because ALD indeed features all the mentioned characteristics. Steatosis, or fatty liver, is characterized by the accumulation of triglycerides in hepatocytes, often seen in early stages of ALD. Pericellular fibrosis is a hallmark of advanced ALD, where fibrous tissue forms around hepatocytes, leading to architectural distortion and cirrhosis. Mallory bodies, also known as Mallory-Denk bodies, are cytoplasmic inclusions composed of intermediate filament proteins, seen in hepatocytes of patients with ALD, particularly in alcoholic hepatitis.

**Why Each Wrong Option is Incorrect**
**Option A:** While fatty changes are indeed a feature of ALD, this option is incomplete, as ALD encompasses more than just steatosis.

**Option B:** Like Option A, pericellular fibrosis is a characteristic of ALD, but this option is also incomplete, as it doesn't mention the other features.

**Option C:** Mallory bodies are a feature of ALD, but similar to Options A and B, this option is incomplete, as it doesn't acknowledge the other characteristics.

**Clinical Pearl / High-Yield Fact**
Early recognition and cessation of alcohol consumption can prevent progression of ALD to cirrhosis. A key clinical correlation is the presence of Mallory bodies in hepatocytes, which can be seen in liver biopsy specimens of patients with ALD, particularly in alcoholic hepatitis.

**Correct Answer Line**
✓ Correct Answer: D. All