Mcq Subject: Pathology

Question 1

In apoptosis, cytochrome C acts through –

A. Apaf 1

B. Bcl-2

C. FADD

D. TNF

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Answer

D. 1 month

Answer

A. Teratocarcinoma

B. Down syndrome

C. Embryonal cell carcinoma

D. Hepatocellular carcinoma

Answer

C. 1 and 3

D. All of the above

Answer

A. Atrophic Lichen Planus

B. Hypertrophic Lichen Planus

C. Bullous Lichen Planus

D. Verrucous Lichen Planus

Answer

A. Ankyrin

B. Alpha spectrin

C. Beta spectrin

Answer

C. Pericardial Fat

D. Peripancreatic fat

Answer

A. Lack of platelet aggregation

B. Lack of reaction accelerator during activation of factor X in coagulation cascade

C. Neutralization of antithrombin III

D. Release of Thromboxane A2

Answer

A. Lack of platelet count

B. Lack of platelet adhesion

C. Lack of factor VIII a during activation of factor X in coagulation coscade

D. Release of thromboxane A2

Question 2

In case of non-hemorrhagic infarct in cerebral coex, reactive astrocytes can be seen as early as ________ after the insult.

A. 1 hr

B. 1 day

C. 1 week

D. 1 month

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Question 3

Alpha fetoprotein is raised in all except:September2011

A. Teratocarcinoma

B. Down syndrome

C. Embryonal cell carcinoma

D. Hepatocellular carcinoma

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Question 4

Organism involved in smooth surface caries is 1. Streptococcus mutans 2. Actinomyces viscosus 3. Lactobacillus 4. Campylobacter

A. only 1

B. only 2

C. 1 and 3

D. All of the above

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Question 5

A 40 year old woman reports with the complaint of burning sensation in the mouth. Clinical examination reveals lesions consisting of radiating white striations in a retiform arrangement affecting buccal mucosa, tongue, lips & gingiva bilaterally. An incisional biopsy is suggestive of lichen planus. The following are different clinical forms of Lichen Planus except

A. Atrophic Lichen Planus

B. Hypertrophic Lichen Planus

C. Bullous Lichen Planus

D. Verrucous Lichen Planus

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Question 6

Most severe form of hereditary spherocytosis is caused by the mutation of –

A. Ankyrin

B. Alpha spectrin

C. Beta spectrin

D. Band 3

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Question 7

Histology of Alport syndrome -a) Foamy cells in interstitiumb) Foamy cells in tubular epithelial cellsc) Thickening of GBM > 100 nmd) Thinning of GBM < 100 nme) Intimal proliferation

A. b

B. c

C. ad

D. ab

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Question 8

In malignant hypeension hyperplastic aeriosclerosis is seen in all except:

A. Hea

B. Kidney

C. Pericardial Fat

D. Peripancreatic fat

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Question 9

Patient with hemophilia A have bleeding disorder because of:

A. Lack of platelet aggregation

B. Lack of reaction accelerator during activation of factor X in coagulation cascade

C. Neutralization of antithrombin III

D. Release of Thromboxane A2

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Question 10

Patient with hemophilia A have bleeding disorder because of

A. Lack of platelet count

B. Lack of platelet adhesion

C. Lack of factor VIII a during activation of factor X in coagulation coscade

D. Release of thromboxane A2

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Mcq Subject: Pathology

In apoptosis, cytochrome C acts through –

In case of non-hemorrhagic infarct in cerebral coex, reactive astrocytes can be seen as early as ________ after the insult.

Alpha fetoprotein is raised in all except:September2011

Organism involved in smooth surface caries is 1. Streptococcus mutans 2. Actinomyces viscosus 3. Lactobacillus 4. Campylobacter

Most severe form of hereditary spherocytosis is caused by the mutation of –

Histology of Alport syndrome -a) Foamy cells in interstitiumb) Foamy cells in tubular epithelial cellsc) Thickening of GBM > 100 nmd) Thinning of GBM < 100 nme) Intimal proliferation

In malignant hypeension hyperplastic aeriosclerosis is seen in all except:

Patient with hemophilia A have bleeding disorder because of:

Patient with hemophilia A have bleeding disorder because of

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